Juvenile Allergic Urethritis with Urethro-vasal Reflex Masquerading Cauda Equina Syndrome

We report a case of severe Juvenile Allergic Urethritis secondary to double concentrate orange squash of a famous brand in a 3-year-old boy who presented with urethral and perineal pain resulting in an abnormal gait and urinary symptoms suggestive of Cauda Equina Syndrome. Ultrasound of the Urinary Tract was normal as was the Magnetic Resonance Imaging (MRI) of the Spine. Withdrawal of the allergen produced complete recovery. Symptoms recurred on food challenge. There are several learning points and take-home messages in this case such as (1) Allergic Urethritis can have a dramatic presentation, mimicking serious conditions such as Cauda Equina Syndrome. (2) Food challenge provided the definitive diagnosis: this is the first report of double concentrate orange squash induced urethritis. (3) Complete avoidance has resulted in an enduring cure. (4) Appropriate timely referral by general Practitioner and cohesive and well-coordinated multidisciplinary team management at the University Teaching Hospital is required to successfully manage such rare and challenging case

Apéndice cutáneo sacro asociado a lipoma del filum terminal y médula anclada: un caso de diagnóstico prenatal

Los disrafismos espinales cerrados tienen una prevalencia aún desconocida e involucran una gran variedad de formas. El lipoma del filum terminal es considerado dentro de los lipomas espinales y suele asociarse a médula anclada. Los estigmas cutáneos lumbosacros no siempre son indicadores de disrafismo espinal cerrado. Reportamos un caso de diagnóstico prenatal de apéndice cutáneo sacro con sospecha de médula anclada, confirmado al nacer como lipoma del filum terminal con médula anclada.

Closed spinal dysraphisms have a still unknown prevalence and involve a wide variety of forms. Lipoma of the filum terminale is considered within spinal lipomas and is usually associated with tethered medulla. Lumbosacral cutaneous stigmata are not always indicative of closed spinal dysraphism. We report a case of prenatal diagnosis of sacral cutaneous appendage with suspected tethered cord, confirmed at birth as lipoma of the filum terminale with tethered cord.

Síndrome de cola de caballo secundario a hernia discal / Cauda equina syndrome secondary to herniated disc / Síndrome de cauda eqüina secundária a hérnia discal

Resumen: Introducción: el síndrome de cola de caballo (SCC) es una entidad poco frecuente, provocado por la compresión de las raíces nerviosas en el canal a nivel de la cola de caballo. Puede dejar graves secuelas si no es diagnosticado y tratado de forma precoz. Únicamente 2% a 6% de las hernias discales lumbares van a provocar un SCC. El diagnóstico de esta patología se basa en criterios clínicos, siendo éstos objeto de controversia dada la variabilidad de presentación del cuadro clínico. El objetivo de este trabajo es analizar la presentación clínica y evolución posoperatoria de los pacientes intervenidos por SCC secundaria a hernia de disco por equipo del Centro de Deformidades de Columna (CE.DEF.CO), entre enero de 2009 y diciembre de 2018. Material y método: realizamos un análisis retrospectivo. La población objetivo son los pacientes intervenidos por SCC secundario a hernia discal entre enero de 2009 y diciembre de 2018 por equipo del CE.DEF.CO. Analizamos 20 pacientes intervenidos quirúrgicamente, de ellos 17 (85%) casos presentaron síntomas urinarios, 18 (90%) casos dolor o elementos deficitarios en miembros inferiores, 13 casos anestesia/hipoestesia en silla de montar, 6 casos síntomas intestinales y 3 casos presentaron disfunciones sexuales. En 19 casos se realizó procedimiento quirúrgico antes de las 48 de iniciados los síntomas. Resultados: la remisión de síntomas esfinterianos al mes es de 83% y al año posoperatorio de casi 87%. Se constató un caso de disfunción sexual al año posoperatorio. Conclusión: el diagnóstico precoz por el médico emergencista es fundamental, por lo que el conocimiento de esta patología es imprescindible. Nuestros resultados en pacientes intervenidos de forma precoz, antes de las 48 horas, fueron similares a los publicados en la bibliografía internacional con bajo porcentaje de secuelas.

Summary: Introduction: cauda equina syndrome (CES) is a rare entity, caused by compression of the nerve roots in the spinal canal at the cauda equina level. It can leave serious sequelae if it is not diagnosed and treated early. Only 2-6% of lumbar disc herniation will cause CES. The diagnosis of this pathology is based on clinical criteria, these being the subject of controversy given the variability of presentation of the clinical picture. Materials and methods: we conducted a retrospective study. The target population are the patients operated on for CES secondary to herniated disc between January 2009 and December 2018 by a team from CE.DEF.CO. (center for spinal deformities). The objective is to carry out a set-up regarding the clinical presentation of this entity and to evaluate the correlation between surgical time and neurological improvement. Out of 20 patients who were operated, 17 (85%) presented urinary symptoms, 18 (90%) were patients in pain or presenting deficit elements in the lower limbs, 13 were cases of anesthesia / hypoesthesia in the saddle, 6 cases evidenced intestinal symptoms and 3 cases presented sexual dysfunction. In 19 cases, a surgical procedure was performed before 48 hours after symptoms started. Results: the remission of sphincter symptoms after one month is 83% and it rises to almost 87% one year after the surgery. One case of sexual dysfunction persisted one year after surgery. Conclusion: early diagnosis by the emergency physician is essential, so knowledge of this pathology is essential. Our experience and good results allow us to conclude that early surgical treatment is associated with symptomatic improvement and fewer neurological sequelae in the short and long term.

Resumo: Introdução: a síndrome da cauda eqüina (SCE) é uma entidade rara causada pela compressão das raízes nervosas no canal no nível da cauda eqüina. Pode deixar seqüelas graves se não for diagnosticada e tratada precocemente. Apenas 2-6% das hérnias do disco lombar causarão a síndrome da cauda eqüina. O diagnóstico desta patologia é baseado em critérios clínicos, sendo este motivo de controvérsia, dada a variabilidade da apresentação do quadro clínico. Materiais e métodos: realizamos um estudo retrospectivo. A população-alvo estava composta por pacientes operados por SCE secundária a hérnia discal entre janeiro de 2009 e dezembro de 2018 pela equipe do CE. DEF.CO (Centro de Defeitos da Coluna Vertebral). Foram analisados 20 pacientes operados, dos quais 17 (85%) apresentaram sintomas urinários, 18 (90%) dor ou elementos de deficiência em membros inferiores, 13 anestesia / hipoestesia em sela, 6 sintomas intestinais e 3 disfunções sexuais. Em 19 casos, um procedimento cirúrgico foi realizado antes de 48 horas após o início dos sintomas. Resultado: a remissão dos sintomas esfincterianos em um mês é de 83% e em um ano pós-operatório é de quase 87%. Um caso de disfunções sexuais foi encontrado um ano após a cirurgia. Conclusão: o diagnóstico precoce pelo médico de emergência é essencial, portanto o conhecimento desta patologia é fundamental. Nossos resultados em pacientes operados precocemente, antes de 48 horas, foram semelhantes aos publicados na literatura internacional com baixo percentual de seqüelas.

Interventional analgesic block in a dog with cauda equina syndrome. Case report / Bloqueio analgésico intervencionista em cão com síndrome da cauda equina. Relato de caso

ABSTRACT BACKGROUND AND OBJECTIVES: The cauda equina syndrome is a neurological condition prevalent in dogs which neurological signs are caused by the compression of the nerve roots located in the lumbosacral spinal canal and is frequently associated with pain, claudication, paresis or paralysis of the hindlimbs and changes in the functioning of the sphincters. The objective of this study was to check the effects of the epidural injection with the combination of dexamethasone, bupivacaine and morphine on the relief of pain and neurological signs in a dog with traumatic cauda equina syndrome. CASE REPORT: Case study of a 2-year old Red Heeler dog, weighing 16kg with a diagnosis of post-trauma cauda equina syndrome. The evaluation consisted of neurological and pain assessment (visual analog scale), quality of life ("5H2M") and infrared thermography. After the initial evaluation and authorization of the tutor, the dog was submitted to general anesthesia and a lumbosacral epidural block, guided by electrostimulation, with the association of dexamethasone, bupivacaine and morphine. After the procedure, the dog showed immediate remission of claudication, paresis and satisfactory analgesia on days 0, 15, 30 and 60 after the intervention. CONCLUSION: The epidural block was effective in improving pain, quality of life and neurological signs and may be an excellent alternative in dogs with pain syndromes associated with the spinal canal.

RESUMO JUSTIFICATIVA E OBJETIVOS: A síndrome da cauda equina é uma afecção neurológica prevalente em cães cujos sinais neurológicos são causados pela compressão de raízes nervosas localizadas no canal espinhal lombossacral sendo frequentemente associada à dor, claudicação, paresia ou paralisia de membros pélvicos e alterações do funcionamento dos esfíncteres. O objetivo deste estudo foi verificar os efeitos da injeção peridural com a associação de dexametasona, bupivacaína e morfina no alívio da dor e dos sinais neurológicos em um cão com síndrome da cauda equina de origem traumática. RELATO DO CASO: Estudo do caso de um animal da espécie canis familiaris, raça red heeler, fêmea, 2 anos de idade e 16kg de peso corporal com diagnóstico de síndrome da cauda equina pós-trauma. A avaliação consistiu no exame neurológico completo, avaliação de dor (escala analógica visual), de qualidade de vida ("5H2M") e por termografia infravermelha. Após a avaliação inicial e autorização do tutor, a cadela foi submetida à anestesia geral e a um bloqueio intervencionista peridural lombossacral, guiado por eletroestimulação, com a associação de dexametasona, bupivacaína e morfina. Após o procedimento, a cadela apresentou imediata remissão da claudicação, da paresia e uma satisfatória analgesia nos dias 0, 15, 30 e 60 após a intervenção. CONCLUSÃO: O bloqueio peridural intervencionista foi eficaz na melhora da dor, da qualidade de vida e dos sinais neurológicos, podendo ser uma excelente alternativa em cães com síndromes dolorosas associadas ao canal espinhal.

Outcome of Delayed Decompression Surgery for Cauda Equina Syndrome secondary to Lumbar Disc Herniation: A Case Report

@#Cauda equina syndrome (CES) is a constellation of symptoms which consist of low back pain, sciatica, saddlearea paraesthesia, urinary or faecal incontinence, with or without motor weakness, and sensory deficit. Surgical decompression is indicated as soon as possible, as decompression within 48 hours from onset allows maximum improvement of symptoms. Recovery usually occurs months or years postoperatively. We report a case of a patient with cauda equina syndrome secondary to massive lumbar disc herniation who had undergone urgent decompression one week after onset of urinary and bowel dysfunction. The clinical outcome post surgery was also discussed.

Cauda Equina Syndrome Occurred by Adhesive Arachnoiditis of the Lumbar Spine with an Unknown Cause / 대한정형외과학회잡지

Spinal adhesive arachnoiditis is an inflammation and fibrosis of the subarachnoid space and pia mater caused by infection, trauma, spinal vascular anomalies, and iatrogenic (surgery and/or puncture). Adhesive arachnoiditis develops various symptoms and signs (gait disturbances, radiating pain, paralysis, and incontinence). On the other hand, adhesive arachnoiditis associated with cauda equina syndrome has not been reported in Korea until now. The authors experienced cauda equina syndrome caused by adhesive arachnoiditis of the lumbar spine with satisfactory results following decompression. We report this case with a review of the relevant literature.

A Good Short-term Outcome in Delayed Decompression of Cauda Equina Syndrome in Klebsiella pneumoniae Spinal Epidural Abscess: A Case Report

Spinal epidural abscess is a severe, generally pyogenic, infection of the epidural space of spinal cord or cauda equina. The swelling caused by the abscess leads to compression or vascular disruption of neurological structures that requires urgent surgical decompression to avoid significant permanent disability. We share a rare case of Klebsiella pneumoniae spinal epidural abscess secondary to haematogenous spread of previous lung infection that presented late at our centre with cauda equina syndrome that showed good short-term outcome in delayed decompression. A 50-year old female presented with one-week history of persistent low back pain with progressively worsening bilateral lower limb weakness for seven days and urinary retention associated with saddle anesthesia of 2-day duration. Magnetic resonance imaging with contrast of the lumbo-sacral region showed an intramuscular collection of abscess at left gluteus maximus and left multifidus muscle with a L3-L5 posteriorly placed extradural lesion enhancing peripherally on contrast, suggestive of epidural abscess that compressed the cauda equina. The pus was drained using the posterior lumbar approach. Tissue and pus culture revealed Klebsiella pneumoniae, suggestive of bacterial infection. The patient made immediate improvement of muscle power over bilateral lower limbs postoperative followed by ability to control micturition and defecation the 4th post-operative day. A good short-term outcome in delayed decompression of cauda equine syndrome is extremely rare. Aggressive surgical decompression combined with antibiotic therapy led to good short-term outcome in this patient despite delayed decompression of more than 48 hours.

Cauda Equina Syndrome by Facet Synovial Pseudocyst Secondary to Epidural Injection / 대한정형외과학회잡지

A 77-year-old woman presented with bilateral leg weakness, accompanied by severe axial back and radicular pain, after a L4–5 epidural injection. She had been receiving misappropriated epidural injections for the last few months. A contrast-enhanced magnetic resonance image showed rim enhancing, spinal canal compromising cystic lesion at the posterior epidural space of L4–5. During surgery, a severely central compromised non-communicating cystic lesion located at posterior epidural space was resected. A histological report of this lesion confirmed a pseudocyst containing a degenerated synovial tissue. Herein, we report our experience of cauda equine syndrome after epidural injection with successful treatment.

Extensive Intradural Epidermoid Cysts with Cauda Equina Syndrome in the Lumbosacral Spine: Case Report

STUDY DESIGN: A case report. OBJECTIVES: To report a rare case of extensive epidermoid cysts in the lumbosacral spine. SUMMARY OF LITERATURE REVIEW: The intradural epidermoid cyst with extensive involvement is rare, and previous reports have reported only extensive intramedullary epidermoid cysts. MATERIALS AND METHODS: A 75-year-old male presented with progressive motor weakness of both extremities beginning 3 days prior. MRI showed extensive intradural extramedullary epidermoid cysts in the lumbosacral region. We performed total laminectomy from the L1 to the L5 level, and the cystic mass was removed. RESULTS: We confirmed the epidermoid cyst on histopathologic examination. CONCLUSIONS: Extensive extramedullary epidermoid cysts are difficult to remove completely. Attempting complete removal may result in neurological deficit. Therefore, when surgical intervention is planned, the poor postoperative prognosis should be taken into consideration.

Huge Intradural Lumbar Disc Herniation Mimicking an Intradural Spinal Tumor: A Case Report

STUDY DESIGN: A case report. OBJECTIVES: To report a case of cauda equine syndrome due to a lumbar intradural disc herniation. SUMMARY OF LITERATURE REVIEW: IDH is rare but there is a higher incidence of neurologic deficit in IDH. Therefore, it should be treated immediately. MATERIALS AND METHODS: A 34-year-old male patient was presented with cauda equina syndrome due to a lumbar intradural mass and underwent surgical excision. RESULTS: Operative findings and the histologic study revealed an intradural disc herniation. CONCLUSIONS: IDH is very rare lesion but should be considered in a differential diagnosis if preoperative MRI demonstrates an intradural lesion.

Cauda Equina Syndrome due to Lumbar Ossification of the Posterior Longitudinal Ligament: A Case Report

STUDY DESIGN: A case report. OBJECTIVES: To report a rare case of cauda equina syndrome due to lumbar ossification of the posterior longitudinal ligament (OPLL). SUMMARY OF LITERATURE REVIEW: Lumbar OPLL with neurologic symptoms is very rare. MATERIALS AND METHODS: A 49-year-old female had experienced weakness in both lower extremities and radiating pain for 1 day prior to presentation. Simple radiography and CT showed OPLL at the L1-L2 level. We performed a total laminectomy and posterolateral fusion at the L1-L2 level using a posterior approach. RESULTS: After treatment, the patient showed improvement of symptoms and is currently living without discomfort. CONCLUSIONS: Cauda equina syndrome due to lumbar OPLL is rare; however, rapid neurologic recovery can be achieved through early diagnosis and surgery.

Neurological Recovery in Two Patients with Cauda Equina Syndrome Secondary to L5 Lumbar Spine Giant Cell Tumour after Treatment with Denosumab without Surgery

We report two patients with cauda equina syndrome (CES) secondary to L5 giant cell tumour (GCT) who achieved good neurological recovery after treatment with denosumab without surgery. The first patient was a 26-year-old man with L5 GCT causing CES who regained bowel and urinary control, muscle power improvement from grade 2 to grade 4 and Oswestry disability index (ODI) improvement from 48 to 23 after denosumab treatment. The second patient was a 25-year-old woman with L5 GCT causing CES who regained bowel and urinary control, muscle power improvement from grade 0 to grade 4 and ODI improvement from 42 to 20 after denosumab treatment. The usage of denosumab in the treatment of patients with CES due to GCT allows potential neurological recovery without any surgical intervention. If surgery is not contraindicated, more time is obtained to prepare the patient preoperatively to attain safer surgery and to achieve complete tumour clearance.

Missed Cauda Equina Syndrome after Burst Fracture of the Lumbar Spine / 대한신경손상학회지

Cauda equina syndrome (CES) is often defined as a complex of symptoms and signs consisting of low back pain, bilateral sciatica, lower extremity weakness, saddle anesthesia, and bowel and bladder dysfunction. CES is considered to be neurosurgical emergency. Delayed or missed diagnosis of CES can result in serious morbidity and neurological sequelae. However, the diagnosis of CES is often difficult when one or more of these symptoms are absent or when these symptoms develop asymmetrically or incompletely. We report a case of urinary retention and sphincter dysfunction without sciatica or motor weakness following an L3 burst fracture in a 52-year-old male and discuss the atypical presentation of CES and treatment of traumatic CES.

Thoracic Spine Tumor Mimicking Cauda Equina Syndrome / 계명의대학술지

Cauda equina syndrome (CES) is one of the typical symptoms condition requires emergency operation. This syndrome is always accompanied by sciatica. Because schwannomas rarely cause sciatica, it is seldom considered as a pain source in the patient with radiculopathy. A 74-year-old male patient presented with lumbar radiculopathy symptoms with, mild stenosis on lumbar MRI scan. All conservative treatment including medication failed to subside the symptoms. Thoracic spine MRI revealed a large tumor in the thoracic region. The symptoms disappeared after excision of the tumor. The tumor was diagnosed as a schwannoma.

Cauda Equina Syndrome Caused by Idiopathic Epidural Lipomatosis

Spinal epidural lipomatosis (SEL) is a rare condition that presents as a back pain with progressive neurologic symptoms. Most affected patients are obese and receiving steroid therapy, or have an endocrinopathies. We report a rare case of cauda equina syndrome caused by SEL in a non-obese healthy young man without any evident traumatic episode. A healthy 19-year-old man, who had experienced lower back pain for two months, visited our emergency room because of the sudden development of motor weakness and voiding difficulty. Lumbar magnetic resonance image revealed extradural fat compressing the cauda equina. Urgent decompression via posterior laminectomy and excision of excess epidural fat resulted in an immediate symptom improvement.

Prognostic Factors of Clinical Outcome of Postoperative Cauda Equina Syndrome

STUDY DESIGN: Retrospective study. OBJECTIVES: We studied the clinical results and prognostic factors for the postoperative caudaequinasyndrome (CES). Summary of Literature Review: The CES is a rare complication, but its aftereffects are serious. And no satisfactory discussion about its accurate treatment guidelines and prognosis has been provided yet. MATERIALS AND METHODS: 10 patients who were diagnosed with a postoperative CES were enrolled from June 2004 to February 2011. Patients were classified into group I with a favorable neurologic prognosis and groupII without neurologic improvement. The medical history, diagnosis, involved segmentand duration till CES was obtained, the duration was performed till second decompression and the clinical symptoms and the outcome of surgical treatment were investigated. RESULTS: Group I contained of 6cases and group 4 of cases.On average were 1.25(0.5-3) hours required for group I and 22(8-38) hours for group II until CES was diagnosed. The time span for the second operation was 7(3-12) hours for group I and 12.25(5-24) hours for group II. Of 6 cases showing motor losswere 4 cases classified as group II at the last follow-up. Of 10 cases with voiding difficulties belonged 4 cases to the group II. Voiding difficulty was continued as clinical symptom in 4 patients of group II after the secondary decompression. CONCLUSION: The less the motor loss and voiding difficulty before the secondary decompression and the faster diagnosis and surgical decompression, the better the prognosis. In particular, as voiding difficulty showed the lowest recovery rate, it is considered to affect prognosis and satisfaction most seriously.

A Case of Cauda Equina Syndrome in Early-Onset Chronic Inflammatory Demyelinating Polyneuropathy Clinically Similar to Charcot-Marie-Tooth Disease Type 1

To present a case of cauda equina syndrome (CES) caused by chronic inflammatory demyelinating polyneuropathy (CIDP) which seemed clinically similar to Charcot-Marie-Tooth disease type1 (CMT1). CIDP is an immune-mediated polyneuropathy, either progressive or relapsing-remitting. It is a non-hereditary disorder characterized by symmetrical motor and sensory deficits. Rarely, spinal nerve roots can be involved, leading to CES by hypertrophic cauda equina. A 34-year-old man presented with low back pain, radicular pain, bilateral lower-extremity weakness, urinary incontinence, and constipation. He had had musculoskeletal deformities, such as hammertoes and pes cavus, since age 10. Lumbar spine magnetic resonance imaging showed diffuse thickening of the cauda equina. Electrophysiological testing showed increased distal latency, conduction blocks, temporal dispersion, and severe nerve conduction velocity slowing (3 m/s). We were not able to find genetic mutations at the PMP 22, MPZ, PRX, and EGR2 genes. The pathologic findings of the sural nerve biopsy revealed thinly myelinated nerve fibers with Schwann cells proliferation. We performed a decompressive laminectomy, intravenous IgG (IV-IgG) and oral steroid. At 1 week after surgery, most of his symptoms showed marked improvements except foot deformities. There was no relapse or aggravation of disease for 3 years. We diagnosed the case as an early-onset CIDP with cauda equine syndrome, whose initial clinical findings were similar to those of CMT1, and successfully managed with decompressive laminectomy, IV-IgG and oral steroid.

Cauda Equina Syndrome Associated with Dural Ectasia in Chronic Anlylosing Spondylitis

Cauda equina syndrome (CES) associated with dural ectasia is a rare neurologic complication in patients with longstanding ankylosing spondylitis (AS). We report a 68-year-old male with a 30-year history of AS who presented a typical symptom and signs of progressive CES, urinary incontinence and neuropathic pain of the lumbosacral radiculopathy. Computed tomography (CT) and magnetic resonance imaging (MRI) findings showed the unique appearances of dural ectasia, multiple dural diverticula, erosion of posterior element of the lumbar spine, tethering of the conus medullaris and adhesion of the lumbosacral nerve roots to the posterior aspect of the dural ectasia. Considering the progressive worsening of the clinical signs, detethering of the conus medullaris through resection of the filum terminale was performed through a limited laminectomy. However, the urinary incontinence did not improve and there was a partial relief of the neuropathic leg pain only. The possible pathogenetic mechanism of CES-AS and the dural ectasia in this patient with longstanding AS are discussed with a literature review.

Spinal Intradural Hematoma and Permanent Paraparesis after a Lumboperitoneal Shunt Operation: An Unusual Complication

Pseudotumor cerebri is a condition of increased intracranial pressure in the absence of clinical, laboratory or radiological pathology. Spinal intradural hematoma formation after lumboperitoneal shunt (LPS) implantation is very rare, but it can cause sudden and serious deterioration. In this report, we present a patient who developed an intradural hematoma following LPS operation. A 27-year-old male patient suffering from headaches and progressive vision loss was diagnosed with pseudotumor cerebri. He underwent LPS operation in January 2009. Four hours after the operation, he developed urinary and fecal incontinence with paraparesis (1/5). Lumbar magnetic resonance imaging identified an intradural hematoma at the level of L2-L3, and he was reoperated. The intradural hematoma was removed. Physical therapy was started because of paraparesis. Two months later, the patient's muscle strength had increased to 3/5. Surgeons must remember that, LPS implantation can cause a spinal intradural hematoma in a small percentage of patients, with catastrophic results.

Spinal Intradural Hematoma and Permanent Paraparesis after a Lumboperitoneal Shunt Operation: An Unusual Complication

Pseudotumor cerebri is a condition of increased intracranial pressure in the absence of clinical, laboratory or radiological pathology. Spinal intradural hematoma formation after lumboperitoneal shunt (LPS) implantation is very rare, but it can cause sudden and serious deterioration. In this report, we present a patient who developed an intradural hematoma following LPS operation. A 27-year-old male patient suffering from headaches and progressive vision loss was diagnosed with pseudotumor cerebri. He underwent LPS operation in January 2009. Four hours after the operation, he developed urinary and fecal incontinence with paraparesis (1/5). Lumbar magnetic resonance imaging identified an intradural hematoma at the level of L2-L3, and he was reoperated. The intradural hematoma was removed. Physical therapy was started because of paraparesis. Two months later, the patient's muscle strength had increased to 3/5. Surgeons must remember that, LPS implantation can cause a spinal intradural hematoma in a small percentage of patients, with catastrophic results.